On Oct 8, 10:32 am, arch...@[EMAIL PROTECTED]
wrote:
> In a recent thread someone was of the view that cows milk was a problem
Having spoken to "someone in a recent thread" whose name (like your
own) shall remain anonymous (not for convenience, clarity or
directness), respecting any need to conceal the identities of the
people discussing milk, while preserving vagueness, I was able to get
a different perspective on your claim adults evolved to drink milk
with no problems.
> because some autoimmune disorders might result
In addition to other disease risks (as stated).
> To which my reply was
> with regard to type 1 diabetes given as an example:
> > As to the other issues you raise, could be, there is no concensus on
it=
..
This vague claim doesn't sup****t your claim adult humans evolved to
drink milk & have no problem metabolizing the contents of milk.
Consensus means most.
Merrium Webster:
Main Entry: con=B7sen=B7sus
Function: noun
Usage: often attributive
Etymology: Latin, from consentire
Date: 1843
1 a: general agreement : unanimity <the consensus of their opinion,
based on re****ts=85from the border =97 John Hersey> b: the judgment
arrived at by most of those concerned
When you do a Pubmed search for "Type 1 diabetes milk", there is a
6/10 relevant item consensus (most sources acknowledge/describe
positive correlations that are biologically plausible between non-
human milk consumption & diabetes, as "someone in a recent thread"
informed you already). Here is a list you'll see with that search:
1. Several studies of infant feeding show a causal relation****p
between time of introduction of formula containing cow protein and
risk of onset of type-1 diabetes mellitus. J Proteome Res. 2008 May;
7(5):2165-7. Epub 2008 Apr 15.
2. Early introduction of cow's milk and cereal, the intake of medicine
during pregnancy, and indoor
smoking of family members are risk factors that predict the
development of prediabetes. Medicina
(Kaunas). 2008;44(1):56-63.
3. No correlation results.
4, 5. =96 NA
6. No correlation results.
7. Analytical epidemiological studies have identified environmental
risk factors operating early in life
which might have contributed to the increasing trend in
incidence=85early introduction of cow's milk
proteins. Pediatr Diabetes. 2007 Oct;8 Suppl 6:6-14.
8. T1D exhibited heightened T-cell reactivities to self-antigens, and
these responses were not strictly limited to the disease target
organs. Children with autoimmune disease and CNS injury also exhibited
abnormal T-cell responses against multiple cow-milk proteins.
Responses to specific milk epitopes distinguished T1D from
inflammatory demyelination and other neurological diseases. Ann
Neurol. 2008 Jan;63(1):98-111.
9. Inverse relation****p (one of your references)
10-12. -NA
10. No correlation results.
14-19. =96NA
20. In humans the primary trigger of insulin-specific immunity is a
modified self-antigen, that is, dietary bovine insulin, which breaks
neonatal tolerance to self-insulin. Ann N Y Acad Sci. 2006 Oct;
1079:350-9.
21. We have shown that exposure to bovine insulin (BI) in cow's milk
(CM) formula induces an insulin-
specific immune response in infants. Pediatr Allergy Immunol.
2006 Nov;17(7):538-43.
So you'll see some variance in associations of milk w/diabetes risk.
The only question is how old does the human offspring have to be and
how much of the diabetes risk is reduced with age while consuming the
cow's milk. Of course the above list is just a fraction of the
hundreds of results. Clicking on "related articles on the right side
of each item will reveal more..
How you are able to use this data to sup****t your claim humans have no
problem metabolizing the contents of milk and actually evolved to
drink milk as adults requires vague claims & switching the subject, as
you've done. Yet Type 1 Diabetes is just 1 of many diseases associated
w/remote suckling of cow udders.
> In which reply a more recent study from 2001 in contrast to the 1994
> first example was offered
2001?
On Oct 6, 8:36 pm, crisology <crisol...@[EMAIL PROTECTED]
> wrote:
> Pediatr Diabetes. 2008 May 21. [Epub ahead of print]
that there is increased risk in those
> genetically "susceptible" to type 1 diabetes.
Those who are not particularly "genetically susceptible" are still at
an increased risk for DM1. Genes are just another risk factor (a
smaller risk factor for most diseases) related to consuming animals.
Until you eat in accordance with genetic script, you'll be more or
less "genetically susceptible" to one disease or another.
This is less then a
> universal for all humans risk even if it proves correct.
I'm not looking for "a universal for all humans" causal agent for a
specific disease. It doesn't exist. Most every disease has a multi
factorial aetiology. I'm looking at unnecessary/increased risks
associated w/various diseases that are preventable with natural diet.
DM1 is just a random disease style that can be earned with foreign
milk consumption that contains difficult/impossible to metabolize
contents for humans.
> Any
> explanation must also account for the great majority of humans who
> consume milk and do not get various autoimmune disorders such as type 1
> diabetes.
Only if the acquired disease has a "universal" causal agent.
Autoimmune diseases do tend to cluster around populations & cancer,
gout, macular degeneration do too. So slow down. We can cover each
disease risk associated with milk. You may survive the DM1 risk but
chol or beta-casein can offer another disease style for you.
> Also as they reviewed the literature they found that gluten and other
> cereal sourced proteins also are related to risk of type 1 diabetes.
"Also."
In addition to cow milk risks, are you "also" risking diabetes (among
other diseases) with cereal too?
> 1: Diabetes Metab Res Rev. 2008 Mar-Apr;24(3):211-22. Related Articles,
Yeah I saw this unique article (# 9) on the search...
> From the discussion section:
>
> This large population-based case-control study in pre-school children
> showed that a long duration of breastfeeding, late introduction of
bottle
> feeding, current cow's milk consumption, higher family's social status,
a=
nd
> a greater number of children in the family were associated with a
reduced
> risk of type 1 diabetes
And I'm questioning/looking for a mode of action for how current cow's
milk consumption is supposed to reduce the risk. Current consumption
as opposed to what? Earlier consumption?
> Recent prospective investigations, which were not subject to recall
bias,
> also produced conflicting evidence on the association between infant
diet
> and the risk of -cell autoimmunity. Some studies did not find an
> association between diabetes autoimmunity and breastfeeding duration or
> early cow's milk introduction [40-43], whereas other studies did
[44][45]=
..
Most studies I found in a random search showed a positive correlation.
Some correlations were more statistically significant. There were
several modes of action described. The link between cow milk &
diseases such as DM1 has been known for a while. It seems lately more
research is being done to investigate it. If "humans have no problem
metabolizing" contents of milk, there would be no issue, yet there are
many diseases related to consuming milk.
> Infant diet has been hypothesized to be involved in the initiation of
the
> type 1 diabetes autoimmune process by impairing the maturation of the
> gut-associated immune system and/or by providing antigens cross-reactive
=
to
> islet cell antigens (molecular mimicry) [22]. However, infant diet may
> affect diabetes risk as well through pathways according to the recently
> raised accelerator hypothesis or the overload hypothesis (e.g. via
> increased birth weight, accelerated growth, increased weight gain) [48],
> because breast-fed children are well known to show less rapid growth and
=
to
> be less likely to develop childhood adiposity than bottle-fed children
> [49]. One study showed both early age at introduction of infant formula
a=
nd
> increased weight gain to be independently associated with increased
> diabetes risk [50].
Yep, another way to obtain diabetes just through milk and although the
gene pool isn't changing much, the disease incidences are. DM1 & 2 are
rising.
> conflicting re****ts
doesn't mean conflicting risk or no consensus but may reflect
different risk factors or incomplete examination. You've only proved
that you'll claim humans have no problem metabolizing cow's milk,
regardless of the risk & regardless of the diseases- and there are
many.
Beyond The Ice Age,
"Someone in a recent thread"
|
